Takahashi M, Abe I, Yoshida N et al. · Nature communications · PMID 42156758
From the original abstract
“These findings establish lymphatic dysfunction as a novel pathogenic mechanism in atrial fibrillation and highlight lymphatic vessel formation as a promising therapeutic target.”
From the closing sentence of the indexed abstract. This is a short quotation from the authors, not a full study appraisal.
Read the full abstract & conclusionCheck the methods for the exact molecule, preparation, and research model. Measurements of a naturally occurring peptide differ from testing its administration. Cell and animal findings need separate evaluation in people.
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